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Laura ThorntonModerator
Like Scott said, I have been co-treating this patient and I cannot stress enough how important the psychiatric referral will be for him. The more that he tells us about his life events and how they coincide with periods of increased pain or injury, this may be one of (if not THE) most important component of treatment. He was hesitant at first to the idea, but seems to be coming around now after this past session.
I don’t want to underestimate the power of manual therapy for this patient either, for both addressing the physical tightness around his cervicothoracic region and hypomobility in both upper cervical and thoracic regions, as well as provide opportunity to discuss pain education, importance of consistent walking program, progressive movement with left arm, and rationale for our other treatments. It has definitely installed more trust from him in us that we are listening to his story and what he believes is helpful for his pain.
I like the ideas of tactile stimulation that you guys have suggested, including the compression shirt and aquatic therapy. I think stressing to him the importance of using his arm for functional tasks will be key for him, like using his left arm for automatic motor patterns like light switches, buttoning shirts, arm swing during gait. He’s a PE teacher and former baseball coach, therefore we might be able to find familiar or automatic movements associated with either that he could practice to assist with plasticity and cortical re-organization.
Laura ThorntonModeratorThanks for sharing this article Nick, and yes, sorry for the very late response.
This diagnostic test reminds me of some of the aspects of external rotation lag sign of the shoulder, minus the proximal stabilization.
I have a hard time believing that a lot of these individuals will be able to achieve the full testing position, especially the 10 degrees of hip extension needed. Hip extension is so commonly lacking in those with hip pathology. I’m not convinced that this test would be more useful over an active movement from neutral into abduction/IR/extension and assessing associated pain during the test.
I do appreciate the added proximal pelvic stabilization as a piece of the test, however I would add it might be beneficial to repeat the test without manual pelvic stabilization, to assess gluteus medius role in both functions.
I would proceed with caution with using this test in isolation for gluteus medius/minimus pathology, at least for physical therapists. There’s no distinction between the spectrum of pathology, from atrophy, tendinopathy, or rupture. The authors claim that based on positive test results, a referral to a surgeon for an endoscopic procedure would be appropriate, which I think is premature, due to limited correlation between pain and disability.
Laura ThorntonModeratorI was thinking the same thing with the Ober test. I haven’t ever seen the Ober test reproduce knee pain, only as a measure of IT band length. I was surprised that they recruited 55 patients who fit the inclusion criteria, but then again it doesn’t state how long the recruiting process was. The authors mention that we can only apply these results to that very specific subgroup of PFPS because it is such a broad group and many, many factors can be at play, I certainly wouldn’t suggest a TFL injection to every anterior knee pain patient that walks through the door.
I think it’s a valid thought in working inhibitory soft tissue to TFL with subsequent gluteus medius strengthening. I was even thinking dry needling as well as an inhibitory method, followed up by gluteus medius strengthening. I do focus a lot on gluteus medius and I tend to catch this pattern of TFL/hip flexor dominance when you do gluteus medius MMT, but I have missed a lot of potential use of inhibitory methods when just purely focusing on facilitating the other glute musculature.
I also agree that the combination of the two interventions was most likely the cause of the 5 year substantial improvement. I have a hard time imagining that the injection alone would have had as big of an impact if they go back to the same mechanics and motor patterns as previously, but then again there was no control group so we can’t say for certain.
That is a really interesting study, thanks for posting! It warrants to keep this in mind when performing the Ober’s test and not forget the TFL as a player.
Laura ThorntonModeratorThanks for posting Nick.
1) Are those two things so mutually exclusive? Isn’t the point of this article to say the exact opposite, that each component should be integrated equally?
2) I understand where you’re coming from with the question, “Is it simply confusing?”. I agree and disagree. The previous framework breaks it down easily, generalizing the three main components into evidence, patient, and expertise. This is a easy concept to learn and teach. But, the authors are correct that it doesn’t include so many important factors like the differences between levels of evidence and including boopsychosocial as an overarching theme. But I feel like a big rationale for this framework is for students and teaching tools, so if you were a new student who was starting to learn this framework to practice, would you get overwhelmed with this new model that they are presenting and need to start with a more basic framework? Its the same with learning levels of evidence. As clinicians who routinely see and practice every day, we more readily can appreciate case reports and detailed studies for our own application. But if you don’t have a reference to relate the case studies to, I understand why the generalized RCTS and SRs are important to begin with. So, I’m playing the neutral card, I understand the importance for both models.
Laura ThorntonModeratorI’m not sure if you would be able to rule out neurodynamic involvement yet, especially with the history of previous strains (with no treatment), possible scar tissue, and close proximity of the structures. If unsure, I would perform a neuro exam too to be sure of any involvement of nerve injuries, and nice idea by Alex for the reverse Slump. If there is a component of neurodynamic involvement, and not addressed, I would think that it might affect overall pain levels, strength gains, and predisposition for further injury, therefore delaying recovery process and ability to progress through exercise program.
Does anyone recommend continuing to monitor SLR/Slump/neuro throughout the recovery process to ensure that scar tissue doesn’t affect nerve mobility?
Laura ThorntonModeratorAlex >> I like Heiderschiet’s article talking about both the theories on effect of eccentrics on scar tissue development:
“An alternative explanation is that the use of early mobilization limits the residual adverse effects of scar tissue formed early in the remodeling process. Early mobilization has been shown to promote collagen penetration and orientation of the regenerating muscle fibers through the scar tissue, as well as re-capillarization of the injured area.”
And the effects in terms of the ability for the muscle to accept more tension due to a shift of the length-tension relationship:
“Scar tissue is stiffer than the contractile tissue it replaces, and thus may alter the muscle-tendon contraction mechanics. Specifically, a decrease in series compliance would shift peak force development to shorter musculotendon lengths. In noninjured subjects, the performance of controlled eccentric strength training exercises has been shown to facilitate a shift in peak force development to longer musculotendon lengths. Therefore, eccentric strength training following a hamstring injury may effectively restore optimum musculotendon length for active tension to normal, thereby reducing the risk of reinjury.”
Laura ThorntonModeratorGood point between tendinopathy vs. strain. There are certainly overlap between both, but there’s a few different important aspects I would consider:
– Lack of inflammatory process with tendinopathy vs. acute tear/strain of hamstring muscle belly including edema, hemorrhage, etc. I would think that strengthening the tendon to allow greater tolerance to load vs. promoting remodeling of muscle fibers would need to be kept in mind.
– Load, Load, Load for tendinopathy. Certainly strengthening is key for both conditions, but I wouldn’t tend do perform manual therapy and other aspects of care that I might add for a strain/tear.
Aspects that overlap: assessment of neurodynamic involvement, agility training, lumbopelvic stability, and progression of resistance exercises incorporating eccentric lengthening when appropriate and pain-free.
Laura ThorntonModeratorTo answer your question on instability features of our patient,
For upper cervical instability features:
– Forward head posture
– Intermittent ringing in the ears
– Minor trauma to neck (surgery)Denies headaches, dizziness, or other cranial/VBI symptoms.
For segmental instability:
– self-manipulation temporarily relieves symptoms until feels “tight” again, especially with prolonged sitting postures.
– location of pain (R U/T)A lot of the research for Grisel’s syndrome are case reports of patients within a very acute period after surgery, with minimal information on long-term results or outcomes. Since she is four months out of surgery, how would this change everyone’s view on evaluation and treatment? I agree with you all that I would certainly continue to monitor all signs/symptoms and proceed cautiously, because of this lack of evidence and potential for serious consequences.
Laura ThorntonModeratorThanks for sharing Oksana! Looking forward to discussing this study at journal club. I think it’s great that you are trying to be as specific as possible with the type of rehabilitation program for him and adding in trunk/pelvis stability training.
You mentioned that you provided him with a verbal cueing of decreasing forward lean during running. What is your rationale for giving him this cue?
I think mapping out a return to run plan would also be an important component to discuss with the patient. The article by Heiderscheit et al from 2010 talks about progressions from stage to stage for hamstring strains, with criteria to progress to return to sport. Without knowing the exact location and extent of the injury, this could help with planning out what he can expect through recovery and keep you guys on the same page. Have you guys discussed a plan for this yet?
Attachments:
You must be logged in to view attached files.Laura ThorntonModeratorI love this article for the references back to hand/wrist positions on the club and effects of ball contact relating back to these injuries/conditions.
The authors also made interesting points about the differences between amateurs and professionals, whereas the professionals tend to have higher swing speeds and greater impact forces, as well as higher tendency for overuse injuries rather than acute.
I would even suggest taking fatigue as a factor as well, where swing mechanics and efficiency would be more impaired at the end of rounds. The same with incline, where wrist positioning at contact might also change when hitting from incline/decline positions. In addition, the concept of hitting “downward” is usually associated with irons, which is different than hitting with a driver in which players would tend to hit up/outward from a tee.
Along with Kyle’s suggestions for treatment approaches, I would suggest a swing analysis with different clubs, incline/decline changes, and possible effect of fatigue to symptoms. This is where I also think communication with the player’s pro/coach would come into play, comparing where and when the player has symptoms to if mechanics or grip positions would need to be modified.
Laura ThorntonModeratorWow, thank you all for responding!
I think it’s only fair to start with updating from our last visit with the patient:
No change in sensation or single heel raise. Can perform them in the pool, but not on land. Reports less uncomfortable sensation, but is getting restless and wants to do more exercise since it’s been several weeks since the onset.
Patient had a EMG with the neurologist he is also seeing: [from patient’s own words, is getting us a copy for next session] “There is definitely something compressing on the nerve, but the nerve is still working somewhat.” Patient has MRI and cortisone injection scheduled (date unknown).
>> Myra, the patient, and I started with a discussion on the research supporting injection + PT has great results compared to each treatment alone. He expressed frustration of the time it was taking to see results, and wondering if surgery is the best option to cut his recovery time down. Myra made some excellent points saying that 1) changing your anatomy forever by laminectomy, etc. to just cut down recovery time isn’t a smart way to look at recovery 2) if the nerve needs time to regenerate and heal (1 mm/day), surgery isn’t going to change that piece. We reassured him on the fact that he is not having any progressive neurological deficits, we will review and start safe and appropriate resistance training at the end of today’s session, and the positive results we are seeing this this type of injury that get better on its own without surgical involvement, it again just takes time.
Lumbar flexion ROM: Fingertips to FLOOR (improved)
Dermatome: Diminished to light touch in the following areas: dorsal and plantar surface of 5th digit, plantar surface of 4th digit, lateral border of foot and lateral 1/3 dorsal and plantar surface of foot, lateral > medial heel, posterior/lateral lower leg up to halfway up lower leg. Posterior/lateral gluteal crease and posterior/lateral thigh. (We wanted to map out entire area to keep close monitoring of sensation)
No change in DTR, no change in strength.
SLR: 90 degrees with posterior/lateral calf “stretch”, worse with dorsiflexion, no change with cervical flexion.
>> Addition of supine sciatic nerve glides with foot in plantarflexion
SLR Reassess: 90 degrees + hip ADD first feeling of “stretch” (worse with dorsiflexion, no change with cervical flexion. Sensation Reassess: no change.
Progress to seated slump sliders with slight thoracic and cervical flexion with full knee flexion >> thoracic and cervical extension and knee extension
SLR Reassess: 90 degrees + hip ADD/IR + dorsiflexion (slight feeling of “stretch”), no change with cervical flexion. Sensation Reassess: no change. Lumbar Flexion AROM: Can reach entire lengths of fingers to floor with slight stretch in posterior/lateral thigh.
Thoracic UPA mobilizations reassessed >> could not reproduce leg pain today, although hypomobility still present in mid-thoracic region. Thoracic mobilizations still performed (with ensuring no reproduction of symptoms during treatment, low grade) as well as lumbar gapping mobilizations to L4-L5.
SLR Reassess: same as previously (no gain or loss, although considering this a full SLR at this point and equal to the other side). Lumbar Flexion Reassess: Can now place entire palm onto floor with slight stretch in posterior/lateral calf.
HEP: Thoracic extensions over ball on wall in standing. Cable column upper extremity resistance including rows, lat pull downs, tricep extensions, bicep curls with ensuring that the anchor stays above shoulder height (to avoid any spinal compression). Addition of front and side planks (this guy can hold 2 ½ minute planks, we were reassessing along entire way for any change in pain, sensation, etc.)
Comments:
It’s definitely a blessing and a curse to treat this patient. He’s young, motivated, fit, and compliant with everything we tell him, but he tends to push the envelope and he wants to get better as fast as possible (can you blame him?) I think it will be essential for us to continue to make sure we are monitoring his neuro status, supporting appropriate exercises he can do, be supportive of the changes he sees, and continue to discuss at each session prognosis.I can’t tell what the prognosis will be in terms of motor function and sensation, or as Myra says there’s no “crystal ball” here. He’s improving with other components (SLR, lumbar flexion, “uncomfortable” sensation in his right leg), which I think is a great sign. I am a huge proponent of reassess and I am on him like a hawk with everything new we add (I’m probably annoying at this point).
There is definitely nerve tension going on within his system with his previous thoracic injuries and Myra sent me to review David Butler’s work on mobilization of the nervous system to review anatomy, pathophysiology, as well as the existence of tension points within our system, one including the mid-thoracic region. I think it’s going to be important to address this component, however I’ve been hesitant until the last session to add any slump neural tension components, because like Nick mentioned, how would you take neurological deficits in mind as you add neurodynamic treatment? Sounds like you all agree to be as slow and gradual as possible, with constant reassess and modifying if he shows increase in irritability or change in neuro status.
Should I take that he can perform a single heel raise in the pool as a good sign? I feel like I’m on the fence, since he wasn’t performing them before. Still cannot perform single heel raise, although can perform double heel raise and has great strength in supine position MMT.
P.S. Unfortunately, I did not consider T4 syndrome in my differential diagnosis list, however it did cross my mind briefly upon reflecting after the session and I did think of you all fondly ha!
Laura ThorntonModeratorThanks for sharing AJ. Great reflection paper. A few things I took away from this article:
Section 3.4.3 >> How many different kinds of touch we provide to the patient! You have never really quantify how much we do touch our patients, not just with specific manual techniques, but with assessment, guidance, demonstration, greeting, goodbyes, etc. It can have significant effects, both peripherally and centrally.
Section 3.4.1 >> “In addition in physiotherapy, the administration of an overt treatment by a mirror feedback was proposed as an effective strategy in chronic LBP (Daffada et al., 2015). Indeed, patients that looked at their back when moving during exercises reported less increase in pain and a faster resolution of their dysfunction (Wand et al., 2012; Diers et al., 2013).”
I need to do this more! I use all the time for retraining in the gym for mechanics and form, but not for placebo effect or to gain a sense of control of the movement. Even encouraging patients to use mirrors at home when performing the exercises could be beneficial. Does anyone use this strategy a lot for this?
Section 3.3 >> There are certainly some ways I can improve on my communication with patients and I admit, when times are busy at the clinic, sometimes adequacy and “this will do” attitude takes over. For instance, when I am writing home exercise programs and I’m in a rush, the sheet won’t give as many specific instructions or I won’t have time to include pictures. It definitely makes me step back and say I need to shape this up because this is the communication that you have with your patient 95% of their time when they aren’t in the clinic.
By the way, I’d be curious to see in person what slanting 90 degrees towards a patient would look like…
Laura ThorntonModeratorWow, that’s fantastic! Nice work.
Do you think that the strength and mobility deficits would be addressed with an independent program and fitness regime? He has returned to work, running, beginning swimming, ADL’s, and is painfree. If he doesn’t require supervision for his program and you don’t think he needs more manual therapy/direct physical therapy, and he feels like he has met all of his functional goals, then is it possible to decrease frequency of visits and see him 1x/month for progression and guidance? If you think that he still requires supervised PT and needs direct treatment, then I would press on. If not, I would begin to transition.
4 months out and we are well into the remodeling phase of healing – which takes time. Dislocation can be a big trauma to the shoulder complex and I wouldn’t be surprised if you see more of a plateau when you get into the later stages of healing. Add in his history of recurrent subluxations of his right shoulder (did we confirm that he has general laxity?), I would think he would need more time to gain motor control and strength but this may be something that he could do on his own without consistent PT sessions.
Laura ThorntonModeratorCan you be more specific on the moderate mobility and strength deficits that he still presents with? Where is he currently in regards to functional return to work, daily activities, running, and swimming?
Laura ThorntonModeratorThe results of this study don’t surprise me. I have difficulty relating the FMS screening movements to the demands that these men and women are subjected to. I like the comprehensive and global movements that comprise the FMS, but to me it’s no wonder why the ability to predict injury is low because they aren’t actually screening the tasks that they need to be able to do.
I am not going to pretend like I know what specific tasks these men and women do every day (my best guesses is running, jumping, endurance training, heavy lifting, shooting, climbing, etc.). I could see how someone who has a really low score (5-9) would warrant further evaluation, but why not do biomechanical screening and analysis with those specific tasks?
“This may suggest that the quality of movement is not the primary indicator of the injury risk and that perhaps other factors such as pain occurrence are more strongly associated with the risk than the actual composite score.” I think this is definitely more of a valid conclusive statement.
Nick >> this was an interesting article on predictors for chronic ankle instability after primary ankle sprain. They speak on different movement tasks and biomechanical faults that might predict sustained instability and repeated injuries:
Recovery From a First-Time Lateral Ankle Sprain and the Predictors of Chronic Ankle Instability: A Prospective Cohort Analysis
Cailbhe Doherty, PhD, Chris Bleakley BSc, PhD, Jay Hertel PhD, ATC, Brian Caulfield, PhD, John Ryan FCEM, FRCSEd, FFSEM, DCH, DipSportsMed, and Eamonn Delahunt, PhD
The American Journal of Sports Medicine 2016; Vol. 44, No. 4: 995-1003 -
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